SLENDERFX
A NOVEL AND PATENTED METHOD
FOR REDUCING BODY FAT

SlenderFX is a safe and natural complex of plant-based polysaccharides and esterified fatty acids that have been shown to reduce stored body fat and enhance weight loss. The improvement in body composition and percent of body fat is achieved through the interaction between the adipose tissue, brain and liver to control serum leptin.

SlenderFX is a unique, proprietary blend that elicits a time-dependent transit through the gastro-intestinal tract. It is this time-dependent transit which elicits the signal to regulate leptin levels. Leptin serves as a regulator of body fat storage by modulating satiation, glycemic control and metabolism. Patented research from the University of Minnesota shows that reductions in serum leptin correlate with lower regional body fat and total body fat. SlenderFX matches up to US Patent No.: 6,899,892 in which the invention provides a method and composition for reducing the level of leptin in the bloodstream and correspondingly reduction in the percentage of body fat. The patent also claims sufficient reductions in the level of serum leptin in the bloodstream to beneficial or efficacious levels in approximately three to six weeks.

SlenderFX also enhances cell membrane stability for improved cellular communication between the liver and adipose tissues for enhanced fatty acid utilization leading to regional fat loss. In addition, modulation of membrane-based inflammatory markers helps to reduce the inflammatory component of fat gain.

SlenderFX containing products from Imagenetix have the attached rights of employing the patent number on labels and brochures. At efficacious levels, suggested structure-function or marketing statements may include the following:

*  Assists in promoting fat loss (Supported by US Patent 6,899,892 and references 1, 2 and 3).

*  Assists in promoting a lower percentage of body fat (Supported by US Patent 6,899,892 and references 1, 2, 3 and 4).

*  Promotes the reduction of stored fat by increasing fatty acid utilization in the fat cell (Supported by references 4 and 5).

*  Promotes the reduction of fat and an enhanced muscle to fat ratio (Supported by references 1, 2, 3 and 4).

*  Promotes the regulation of energy balance allowing for improved mental energy (Supported by references 6 and 7).

 

 

 

1. Eikelis N and Esler M. The neurobiology of human obesity. Exp Physiol. 2005; (90)5: 673 – 682.

2. Drevon CA. Fatty acids and expression of adipokines. Bichimica et Biophysica Acta 2005, 1740: 287-292.

3. Meier U, Gressner AM. Endocrine regulation of energy metabolism: Review of pathobiochemical and clinical chemical aspects of leptin, ghrelin, adiponectin and resistin. Clin Chemistry 2004, 50(9): 1511-1525.

4. Cohen P, Miyazaki M, Socci ND, Hagge-Greenberg A, Liedtke W, Soukas AA, Sharma R, Hudgins LC, Ntambi JM, Friedman JM. Role for stearoyl-CoA desaturase-1 in leptin-mediated weight loss. Science. 2002 12; 297(5579):240-3.

5. Cohen P, Friedman JM. Leptin and the control of metabolism: role for stearoyl-CoA desaturase-1 (SCD-1). J Nutr. 2004 Sep; 134(9):2455S-2463S.

6. Greenwood CE, Winocur G. High-fat diets, insulin resistance and declining cognitive function. Neurobiol Aging. 2005 Oct 27.

7. Elias MF, Elias PK, Sullivan LM, Wolf PA, D'Agostino RB. Obesity, diabetes and cognitive deficit: The Framingham Heart Study. Neurobiol Aging. 2005 Oct 10.

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